Pretty simple.
You want to lubricate the satiation receptors with good fat.
Vegetable lecithin is a satiator and when consumed before a meal lowers one's consumption.
Therefore if one uses logic the steady state OF vegetable oils lining the gut may deter
one from eating much at all.
It seems when one **lines** the gut / small intestine with oil .. the
length of **time** the gut wall is exposed TO the oil is the .. KEY ..
to the satiation / "no thanks I am not hungry.".
"Extent of Spread of Lipolytic Products Along Small Bowel"
Role of small intestine in caloric compensations to oil premeals in
rats
Am J Physiol Regul Integr Comp Physiol 275: R1320-R1333, 1998;
Vol. 275, Issue 4, R1320-R1333, October 1998
J. H. Meyer, M. Hlinka, A. Khatibi, H. E. Raybould, and P. Tso
Departments of Physiology and Medicine, University of California Los
Angeles School of Medicine and the West Los Angeles and Sepulveda
Veterans Affairs Medical Centers, Los Angeles, California 90073; and
Department of Physiology, Louisiana State Medical School, Shrevesport,
Louisiana 71103
We postulated that dose-responsive satiety after oil premeals varies
with the number of gut sensors stimulated by lipolytic products along
intestine. These experiments in fasted rats on satiety after oil
premeals were performed to 1) determine whether satiety was induced by
lipolytic products but not triglycerides; 2) confirm that oil empties
from the stomach at rates that vary with oil loads; 3) ascertain that
increasing rates of oil entry into duodenum extend the length of gut
contacted by lipolytic products; and 4) judge whether length of gut
contacted correlated with dose-responsive satieties to dietary oils.
5) Using specific antagonists, we attempted to define how satiety was
signalled by gut sensors. Timing and degrees of satiety did not
correlate with timing and extent of gastric distensions but, rather,
with the timing and extent of spread of lipolytic products along small
bowel. Satiety after the highest premeal load of oil was blocked by
Pluronic L-81, an inhibitor of intestinal secretion of apolipoprotein
A-IV, but was unaffected by MK-329 (a specific antagonist of
cholecystokinin) or by capsaicin blockade of chemosensory nerves.
caloric adjustments; temporality
Herbivore Hypothesis
http://sites.google.com/site/herbivorehypothesis/age-related-iron-accumulation